I wanted to give an update on my “Possible replacements for Delgado” post. Mets’ first baseman, Carlos Delgado was placed on the 15 Day DL and had hip surgery on Tuesday. Mets’ GM, Omar Minaya expects Delgado to be out about 10 weeks.
It appears the Mets will go with 2 of the internal options that I had on my list of replacements: Fernando Tatis and Daniel Murphy. The Mets keep asking a lot of Murphy. In the last year and a half, the Mets have asked Murphy to learn how to play LF, 2B and 1B. That is a lot to ask of a young player who is trying to get accustomed to the major leagues.
In terms of trades, here is what Minaya had to say…
“I’m never going to stop looking around,” Minaya said. “We’re always involved in talking to clubs.”
A name that has been mentioned as a possible trade candidate that I didn’t think of is New Jersey product, Mark DeRosa. DeRosa would be a perfect fit for the Mets. He can play LF, 1B, 2B, RF and 3B just in case they want to give Wright a break down the stretch.
One last update from this post. In the post, I asked the question “Seriously, what is going on with all the hip injuries lately? Utley, Arod, Lowell, Gordon, etc…Has someone looked into this? It’s crazy”
One of my readers John from Omaha, NE has offered his opinion on why there are so many hip injuries lately. Pretty interesting….
I have a theory on the rash of baseball hip injuries. Steroids.
My wife has asthma. She has had frequent hospital stays and has been treated with steroids for over 7 years. She developed pain in her hip, and was diagnoised with avascular necrosis I cut and paste from Wiki the symptons and cause. To me, this growing phenomena of baseball players developing hip injuries is going from a coincidence to a trend.
Avascular necrosis (also osteonecrosis, aseptic (bone) necrosis, ischemic bone necrosis, and AVN) is a disease resulting from the temporary or permanent loss of the blood supply to an area of bone. Without blood, the bone tissue dies and the bone collapses.If avascular necrosis involves the bones of a joint, it often leads to destruction of the joint articular surfaces (see Osteochondritis dissecans).
There are many theories about what causes avascular necrosis. Proposed risk factors include alcoholism, excessive steroid use, caisson disease (decompression sickness), vascular compression, hypertension, vasculitis, thrombosis, damage from radiation, bisphosphonates (particularly the mandible), sickle cell anaemia, and Gaucher’s Disease. In some cases it is idiopathic (no cause is found).Rheumatoid arthritis and lupus are also common causes of AVN. Prolonged, repeated exposure to high pressures (as experienced by commercial and military divers) has been linked to AVN, though the relationship is not well-understood. post trauma,”
Good work John!!! As always, we here at The Ghost of Moonlight Graham value people’s opinions. What does everyone else think? Opinions and thoughts are welcome.